RESEARCH ON OBSESSIVE COMPULSIVE DISORDER / TOURRETTE’S
With Obsessive Compulsive Disorder (OCD), a person can’t stop repeating specific behaviors or stop his or her brain from repeating particular thoughts. A substantial body of research shows that problems with OCD are related to the functioning of areas in the front of the brain. If that part of the brain is working too slowly or quickly, a person is unable to stop repeating certain thoughts or behaviors.
Many therapists and other health professionals using neurofeedback to treat OCD note marked reductions in OCD symptoms in their clients after neurofeedback training. People with OCD relate that, after neurofeedback training, they do not really need to make an effort to stop unwanted repetitive thoughts and behaviors. They say that they their minds are much quieter. With neurofeedback training, the brain learns to respond to situations in a more conventional and healthy manner.
CASE STUDIES ON OBSESSIVE COMPULSIVE DISORDER / TOURRETTE’S
A Case Series [pdf]
Tanju Siirmeli and Ayben Exrteme
Clinical EEG & Neuroscience, Volume 42 No 3
While neurofeedback has been extensively studied in the treatment of many disorders, there have been only three published reports, by D.C. Hammond, on its clinical effects in the treatment of obsessive compulsive disorder (OCD). In this paper the efficacy of QEEG-guided neurofeedback for subjects with OCD was studied as a case series. The goal was to examine the clinical course of the OCD symptoms and assess the efficacy of QEEG guided neurofeedback training on clinical outcome measures. Thirty-six drug resistant subjects with OCD were assigned to 9-84sessions of QEEG-guided neurofeedback treatment. Daily sessions lasted 60minutes where 2 sessions with half-hour applications with a 30 minute rest given between sessions were conducted per day. Thirty-three outof36 subjects who received neurofeedback training showed clinical improvement according to the Yale-Brown obsessive-compulsive scale (Y-BOCS). The Minnesota multiphasic inventory(MMPI) was ad-ministered before and after treatment to 17 of the subjects. The MMPI results showed significant improvements not only in OCD measures, but all of the MMP1 scores showed a general decrease. Finally ,according to the physicians’ evaluation of the subjects using the clinical global impression scale (CGI), 33 of the 36 subjects were rated as improved. Thirty-six of the subjects were followed for an average of 26months after completing the study. According to follow-up interviews conducted with them and/or their family members 19of the subjects maintained the improvements in their OCD symptoms. This study provides good evidence for the efficacy of neurofeedback treatment in OCD. The results of this study encourage further controlled research in this area.
Neurofeedback with Obsessive-Compulsive Disorder [pdf]
D. Corydon Hammond, PhD
Professor, University of Utah School of Medicine, Salt Lake City, Utah
Obsessive-Compulsive Disorder (OCD) is often less than optimally treated using medication or behavior therapy. However, qEEG and neuroimaging research have identified brain patterns associated with OCD (Prichep et al., 1993).
Two patients with OCD were screened with the Padua Inventory, the Yale-Brown Obsessive-Compulsive Scale, qEEG, and in one case, the MMPI. Each patient displayed different qEEG patterns associated with OCD. Neurofeedback individualized to qEEG findings was used.
A simple and a complex tic (Gilles de la Tourette’s syndrome): their response to EEG sensorimotor rhythm biofeedback training. [link]
This study presents a clinical treatment regime for the treatment of tic manifestation, both simple and complex. The response of a case of simple tic and a case of complex tic (Gilles de la Tourette’s syndrome) to EEG sensorimotor rhythm biofeedback training are presented. Specifically, the simple and the complex tic, both of long duration, were eliminated via this EEG biofeedback training procedure. It is hypothesized that this exercising of the sensorimotor cortex resulted in increased activation of this cerebrocortical subsystem and was reflected in increased voluntary muscle control and a heightened threshold for random motor discharge, resulting in the elimination of both tics as in the response of cases of epilepsy with motor involvement to EEG sensorimotor rhythm biofeedback training. The additional psychophysiologic sequelae of the complex tic–attention deficit disorder–remediated in the manner of the response of learning-disabled to EEG sensorimotor rhythm biofeedback training.